Question: What is the detailed step-by-step PTEN signaling pathway? Discuss and support your analysis with cited references and data from published scientific articles.
This question must be answered in the Introduction.
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Provide a title for your lab report.
Author:
Your full name and mentor(s).
Significance:
Detailed paragraph (i.e. multiple sentences describing the summary of previously published research.
Introduction:
Provide detailed background information on your research topic while citing relevant references (scientific publications only, about 15 citations). Briefly state the major question of lab project, reiterate your hypothesized pathway, and describe how you will go about addressing the hypothesis. This section will involve multiple paragraphs.
Materials and Methods:
Write the details of your reviewed literature, data analysis, all the resources and databases used in the meta-analysis.
Results:
Provide brief description of the data.
Discussion:
Discuss the results of your review especially in relation to your hypothesized pathway.
Conclusion:
Provide a summary conclusion of your findings.
Acknowledgement:
Provide any support in terms of personnel, resources, and funds.
References:
Provide a formatted list of your curated citations relevant to your research as a bibliography (scientific publications only, greater than 15 citations) .
The PTEN gene is deleted in multiple types of advanced human cancers. The resulting loss of the PTEN protein contributes to the uncontrolled growth of cells. Indeed, cells lacking PTEN have elevated levels PI(3,4,5)P3 and PKB activity. Because PKB exerts an anti-apoptotic effect, loss of PTEN reduces the programmed cell death that is the normal fate of many cells. In certain cells, such as neuronal stem cells, absence of PTEN not only prevents apoptosis, but also leads to stimulation of cell cycle progression and an enhanced rate of cell proliferation. Knockout mice lacking PTEN have big brains with an excess numbers of neurons, attesting to PTEN’s importance in the control of normal development.
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